Heel Therapeutic Index

Heel Therapeutic Index

Classical homeopathy according to Hahnemann (1811) orients itself based on the so-called ‘drug picture’ to determine the appropriate remedy. It claims that symptoms of disease behave reciprocally towards those symptoms which the healthy test person develops after  the intake of a mother tincture or a diluted substance (potency.) The principle of action which can be derived thereof is known as the Simile Principle (Similia similibus curentur = Likes may be cured by likes.) The clinical syndrome occurring in a patient can be overcome by an artificially induced, similar disease. The Aequalia Principle (Isopathy = the condition may be healed by the causative substance) can also be applied with potentised allopathic substances or partly with nosode preparations in anti-homotoxic medicine and/or with vaccines in conventional medicine.

Classical homoeopathy works with single remedies which are only partly truly single- constituent remedies, (e.g., sulphur, mercury, arsenic, etc.), or which are otherwise botanical extractions containing a highly complex mixture of numerous constituents. Repertories (lists of symptoms produced by drugs) facilitate the selection of the most appropriate remedy in homoeopathy.

Anti-homotoxic medicine usually pursues an indication-oriented approach. The anti- homotoxic remedies predominantly represent mixtures of substances of low to middle potencies. Through practical application in homoeopathy it became obvious that the use of concentrated or poisonous tinctures could damage the patient and that, therefore, they could only be used in homoeopathic dilutions, i.e., potencies. This practice was scientifically supported by Rudolf Arndt (psychiatrist, 1835-1900) and Hugo Schulz (pharmacologist, 1853- 1932) through a quantitative differentiation of the medicinal effect on bio-systems and still applies as the Arndt-Schulz Principle. It states:

  • weak stimuli stimulate the life functions (retro-action of homoeopathic preparations)
  • moderately strong stimuli accelerate them
  • strong stimuli act as inhibitors
  • the strongest stimuli suspend the life functions

Since several tissue-incompatible substances are usually involved during the development of a disease, the simultaneous use of several potentised ”antitoxins“, as present in the anti- homotoxic preparations, is justified.

Against the background of the conflicting medicinal and therapeutic concepts promulgated in humoral pathology, cellular pathology, molecular pathology, and related fields including modern cybernetics, the German physician Dr. Hans-Heinrich Reckeweg formulated Homotoxicology in 1952. This conception was developed from homoeopathy for the purpose of providing a holistic perspective on the synthesis of medical science.

Reckeweg formulated an essential tenet of Homotoxicology, as follows:

”According to Homotoxicology all of those processes, syndromes, and manifestations, which we designate as diseases, are the expression thereof that the body is combating poisons and that it wants to neutralize and excrete these poisons. The body either wins or loses the fight thereby. Those processes, which we designate as diseases, are always biological, that is natural teleological processes, which serve poison defence and detoxification.“

Referring to conventional medical indications connects anti-homotoxic medicine with allopathy, while therapy with potentised substances unites it to homoeopathy. Anti-homotoxic medicine is the connecting link between allopathic medicine and homoeopathy.

Fig. 1
Conventional medicine:

The higher the concentration, the stronger the effect (dose-effect relation; increase of side effects).

Homoeopathy:

Increase of the effect with decreasing concentration (effect optimum not definable). Anti-homotoxic medicine:

Connecting link between conventional medicine and homoeopathy.

Homotoxins

Homotoxins are all of those substances (chemical/ biochemical) and non-material influences (physical, psychical), which can cause ill health in humans. Their appearance results in regulation disorders in the organism. Every illness is due therefore to the effects of homotoxins. Homotoxins can be introduced from the exterior (exogenic homotoxins) or originate in the body itself (endogenic homotoxins).

Homotoxons

These are understood as chemical reaction products from compounds of homotoxins with each other or with other substances (e.g., products of metabolism) which neutralize the poisonous property of the homotoxins. The best example thereof is the liver, in whose cells homotoxins and metabolic products are united to detoxify the organism.

Retoxins

Deposits of homotoxins with endogenic substances, which cannot be eliminated via excretion or irritation, are designated as ”residual poisons“ (retoxins). The most important example thereof is the non-enzymatic glucosilisation of tissues and cell surfaces in case of glucose excess, as with, among others, latent diabetes mellitus.

Homotoxicosis – The Concept of Disease in Homotoxicology

Homotoxicosis is a non-physiological condition which arises after reaction of a homotoxin on cells and tissues. A homotoxicosis occurs as a humoral or cellular appearance and can be followed by morphological changes on tissues. The homotoxicosis is named after the homotoxin which triggers it. The homotoxicosis leads to defensive measures of the organism whose goal is to eliminate the homotoxins and to restore the physiological conditions when possible.

The Ground Regulation

This refers to the local regulation possibilities of the ground system along with its superimposed nervous, hormonal, and humoral regulation systems. The ground system is composed of the ground substance plus cellular, humoral, and nervous components. The ground substance (extracellular matrix) is formed of highly polymerised sugars  (proteoglycans and glycosaminoglycans) plus structural and meshing glycoproteins.

The Phase theory

The Six-Phase-Table illustrates the chronological courses of various symptoms of a disease within the framework of the ground regulation. The single phases are transient into each  other and demonstrate typical phasal indicating signs. The Six- Phase-Table is subdivided into three sections (humoral phases, matrix phases, cellular phases), each of which is subdivided into 2 phases. Two phases are allocated to the excretion principle (phases 1 + 2), the deposition principle (phases 3 + 4), and the degeneration and/or deterioration principle (phases 5 + 6). The Biological Division runs within the matrix phases.

Fig. 2: The Six-Phase-Table
The humoral phases

In the humoral phases the intracellular systems are not disturbed. The defence system is intact and can excrete the homotoxins via various paths.

Excretion PhaseThis phase contains manifestations of increased physiological excretion mechanisms.
Reaction PhaseIllnesses of this phase are marked by an exudative inflammation, which enables an accelerated excretion of toxins from the body.  
The matrix phases

In these phases the homotoxins are deposited at first in the mesh of the extracellular matrix. During the further course its structural components as well as functions are altered. In case of continuing illness increasing stress and damage of the intracellular structures result.

Deposition PhaseIn this phase the excretion mechanisms of the body are overworked and toxins are deposited in the matrix. This phase often progresses with few symptoms.  
Impregnation PhaseDiseases in this phase are characterized by the presence phase of toxins which become a part of the connective tissue and the matrix, along with changes in the structural com- ponents as well as their functions. The typically increasingly severe symptoms and signs of this phase demonstrate damage of the organ cells.  
The cellular phases

During the cellular phases of a disease, cell systems are increasingly destroyed. The defence system is no longer able to excrete the toxins out of the cells or out of the matrix by virtue of its own strength. Typical for these phases is the so-called regulation rigidity.

Degeneration PhaseDuring this phase, courses of disease cause serious damage, and destruction of larger cell groups of an organ takes place.
Dedifferentiation (neoplasm) PhaseDiseases of this phase are characterized by the development of undifferentiated, non-specialized cell forms. Malignant diseases stand at the end of this phase.

The Biological Division refers to the imaginary boundary between the deposition and impregnation phases. It demarcates the pure deposition in the matrix from the integration of toxins into its structural components. Whereas a simple excretion of the toxins is possible during the deposition phase, structural and functional changes are found in the impregnation phase. Thus the spontaneous endogenic excretion of the homotoxins is impeded.

The term ”vicariation“ refers to the transition of the indicating signs of an illness within one phase to another organ system, or the change of the fundamental symptoms and signs into another phase, with or without a change of the organ system.

Progressive vicariation: Progressive vicariation refers to an aggravation of the total  symptoms and signs of illness.

Regressive vicariation: Regressive vicariation refers to an improvement of the total symptoms and signs of an illness.

The different components of the anti-homotoxic preparations activate the defense system of the body. The immune system with its memory and regulation systems can be compared to the spiritual-mental self, the ego. The deposition phase and more frequently the impregnation phase are characterized by immunological processes such as chronic inflammation and auto-aggression. The humoral area (via immunoglobulins from B-lymphocytes) and the cellular area (T-cells, granulocytes, macrophages) still counterbalance each other in this case. A regressive vicariation is still possible in these matrix phases.

Herein lies a great opportunity for anti-homotoxic medicine. The immunological bystander reaction represents a theory of anti-homotoxic therapy for inflammatory illnesses. It   is based on low dose antigen reactions, particularly of substance combinations in the range of 1X to 14X, with 4X to 8X appearing to be the most favorable. The bystander effect cannot be triggered at higher potencies. Experience shows that higher potencies as well as trace elements and intermediary catalysts are able to stimulate the ground regulation. (Functional circle of anti-homotoxic therapy; Heine 1997.) It is significant that for the potency ranges 3X to 12X, a considerable difference exists in the activation of specific enzyme systems compared to substances conventionally diluted to the same concentration. The dose-action relationship of potentized substances compared to diluted substances demonstrates a non-linear relationship.

Every organism requires energy to maintain its vital functions which must be continuously provided by the metabolism. Disorders of the energy metabolism, therefore, impair the energy supply which is controlled by endogenic regulation. The organism is an energetically open system for which suitable energy, in the form of food, must be supplied and unsuitable energy must be evacuated. In this manner an unstable state of order can be maintained, far from a thermodynamic balance, for a longer period of time; a “life- span.” All reactions of the organism proceed at relatively low temperatures in the aqueous milieu and must be accelerated, i.e., catalyzed. The prerequisite for an effective catalysis is suitable substrates between and in the cells. Because the extracellular space is located in front of the cells, the cells can only react as they have been informed via the extracellular space. The dynamic structure of the extracellular space and its regulation (ground regulation) have therefore a decisive impact on the effectiveness of the extracellular and intracellular catalysts. This depends on the structure of the ground substance (extracellular matrix or simply, matrix). It forms in all cells and cell groups a molecular sieve of matrix components such as highly polymerized sugar protein complexes and sugar complexes (proteoglycans-glycosaminoglycans, PG/GAG’s), structural proteins (collagen, elastin) and meshing glycoproteins (e.g., fibronectin) (Figure 2). As the PG/GAG’s are electro-negatively charged, they are able to bind water and exchange ions as well. They are therefore the guarantors for isoiony, isoosmy and isotony in the matrix.

Figure 2: Diagram of the ground regulation system
Reciprocal relationships (arrows) between capillary system (capillaries, lymph vessels)

ground substance, terminal autonomic axons, connective tissue cells (mast cells, defense cells, fibroblasts, etc.) and parenchyma cells. Epithelial and endothelial cell groups rest on a basal membrane which mediates to the ground substance. Every cell surface carries a glycoprotein and glycolipid film connected to the ground substance (dotted line), to which the histocompatibility complexes (MHC) also belong. The ground substance is connected to the endocrine system via the capillary system and via the axons to the central nervous system. The fibroblast is the metabolically active center (Heine, 1997.)

The connection to the central nervous system is conducted via the autonomic nerve fibers blindly ending in the matrix. The connection to the system of endocrine glands (pituitary gland, thyroid gland, suprarenal gland, etc.) is conducted via the capillary system which permeates the matrix. Both systems are connected to each other in the brainstem and to superimposed centers of the brain. In this manner, the matrix is regulated not only on site but also under the influence of superimposed control areas. The regulation center in the matrix is the fibroblast (corresponding to the glia cell in the central nervous system). It reacts immediately to all incoming information (hormones, neural substances, metabolites, catabolites, pH value changes, etc.) with a synthesis of ‘good’ or ‘bad’ information. In this manner every surplus or deficit can lead in certain circumstances to detrimental consequences for the total system depending individually on a vicious cycle.

It is important to note that due to the sieve-like as well as connective properties of the PG/ GAGs, the danger of damaging the matrix also exists through the development of a latent tissue acidity, increase of free radicals and the activation of the proteolytic system turning into a pro-inflammatory situation. Ultimately, damage of all humoral and cellular elements may arise progressing at first from persistent feelings of ill health to chronic disease and malignant processes (Heine, 1997.)

Homotoxicological remedies have the ability to achieve immunomodulation. This immunomodulation is achieved via ingredients in the remedies used; these remedies contain an amino acid sequence in dilution, therefore remedies with plant extracts, suis organ extracts, nosodes and animal venoms are able to immunomodulate. The critical dilution of the ingredients from 1X-14X, furthermore gives the remedies the ability to induce the immunological bystander reaction, primarily in the Gut Associated Lymphoid Tissue (GALT) and less so in the peripheral tissues.

The normal immune response depends, apart from the non-specific immune response via the phagocytes, complement and acute phase proteins, on the specific immune response, mediated by the lymphocytes and their products.

The CD4, CD8 and the family of Large Granular Lymphocytes (Including the Natural Killer Cells) forms part of this normal arm of the immune response. The CD4 and CD8 cells need to be activated by an Antigen Presenting Cell (APC) which will present the antigen by processing it either via the Major Histocompatibility Complex I or II (MHC I or II) or via other molecules such as the CD1 molecule in the intestinal epithelial cell. MHC I will activate CD8 cytotoxic cells, MHC II the CD4 helper cells and CD1 from the Intestinal Epithelial Cells (IECs) will activate CD8 suppressor cells.

CD4 cells have several subclasses of functional cells, namely Th1, Th2, Th3 and also Th4 cells. Th1 cells secrete the cytokines TNF and Interferon gamma and will activate lymphocytes and products which have significance within cellular immunity. Under pathological conditions, Th1 cells will cause diseases like Rheumatoid arthritis, Inflammatory bowel disease and other organ related autoimmune diseases such as Multiple sclerosis. It is also responsible for the general inflammatory processes we see in the tissues in general. Th2 cells on the other hand secrete the cytokines IL-4, 10, 5, and 13 and have significance within antibody formation. Th2 cells are also the pathway through which allergic phenomena are mediated in pathological circumstances. In the normal organism, there is a constant oscillation between Th0, Th1 and Th2 cells and this oscillation has a chronobiology similar to most of our other regulatory systems. In factTh1/Th2 oscillation is under the control of the neuroendocrine system, and will follow its diurnal rhythm. Another mechanism which achieves this Th1/Th2 oscillation is the inhibitory effect the Th1 and Th2 secreted cytokines will have on each other. Another cell which can also modulate the normal oscillation is the Th3 cell.



This cell secretes the cytokine Transforming Growth Factor beta, (TGF-ß) and can modulate the balance between Th1/Th2. In addition the Th3 cell and certain CD8 suppressor cells are most likely responsible for the development of our immune tolerance to foods. It seems that the size of the antigen which is presented by the APC will determine whether it will generate a Th1, Th2 or Th3 response. According to Heine, any substance in the dilution from 1X-14X will have the effect of generating a Th3 response, and to a lesser extent, a Th2 response.

This refers, as its name implies, to the induction of cells by a relative non-toxic antigen which will then have an effect on a process such as an inflammation which was started by another antigen somewhere else in the body. Let’s look at the bystander reaction (Figure 3).

Figure 3: The bystander reaction

In order to initiate the bystander reaction we need a small dose of antigen, or a remedy containing plant extract, suis organ extract, animal venom or a nosode, each in a specific dilution. These diluted substances are then taken up by the APC’s in the gut lining which will present them as an antigen to the lymphocytes in the GALT via the MCH or the CD1 binding mentioned above. The processed antigen is then displayed as a motif on the outside of the APC. There will be a motif for each of the ingredients, thus if we give Traumeel, one for Arnica, one for Bellis perennis, etc., or if we give Hepar compositum, one for Hepar Suis, one for Colon Suis and so forth.

After being presented with the motifs on the APC, prolymphocytes of the Th3 type are activated due to the size of the motif (5-15 amino acids), and will differentiate into a population of Th3 cells. There will be a Th3 type cell for each of the motifs presented, thus one for Arnica, Bellis perennis and so forth.

These lymphocytes then migrate to the lymph nodes in the Peyer’s patches and proliferate into billions of copies. This process is called cloning.

The millions of cloned Th3 cells will then through chemotaxis find the inflammation caused by Th1, Th2 and Th4 lymphocytes and suppress them, thereby restoring the balance and normal oscillation between Th1/Th2 cells. Here the law of similars is at work, as each Th3 cell for Arnica or Bellis perennis for instance, will find the corresponding Th1/Th2 cell which looks similar to its motif and suppress it.

The immunological bystander reaction serves as a model for the working mechanism of immunomodulatory remedies which, through a non-toxic substance in a specific dilution can induce a regulatory cell, the Th3 cell . This regulatory cell, while developed at another site, and acting as an innocent bystander can bring about a balance in the immune system, by down-regulating an inflammatory process started by another noxious homotoxin remote from its own activation. The Th3 cell acts as a fulcrum on which the balance of Th1 and Th2 cells rests (Figure 4).

This also confirms one of the ways which Dr. Reckeweg postulated as to how homotoxicological remedies work. He theorized that by giving a non-toxic substance the defense system of the body is activated through another pathway in order to bring about homeostasis.

Figure 4: Diagram of Th3 as a fulcrum – Due to the dilution and ingredients found in certain homotoxioclogical remedies they lend themselves well to this type of immunoregulation.

Homotoxicological medicines have been developed to serve the very specific purpose of restoring regulation in the organism. Although they may appear as being complex homeopathic compositions, they in fact differ in a number of ways from the normal complex prescription, in that they incorporate various dilutions of remedies, as well as potentized catalysts, allopathics and vitamins, for instance. As our knowledge of molecular biology increases we can witness the deep-acting function of these combinations on the body. We classify the remedies according to the origin of the remedy, the combination, as well as the phase in which they will be useful.

The remedies are made up of four pharmacological groups (PG’s):

  1. The first group is the plant materials (PPG’s). Depending on the dilution, these could have either the dominant, secondary or complementary action in the remedy.
  2. The second group is the mineral pharmacological group or MPG’s. In the potency they mostly appear in the remedies, they act as functional regulators, modifiers and stimulators.
  3. The third group is unique to Homotoxicology, and is made up of the catalysts (CPG’s). These are mostly in the 6X-10X dilution range, and will act as modulators and regulators. The CPG’s may contain potentized elements of the Krebs cycle, and carrier enzymes of the respiratory chain such as Ubichinon (Coenzyme Q10). Hormones in potentized form, as levothyroxin and cortisone, can also act as catalysts. Certain other substances including Procainum, Glyoxal and Methyl glyoxal will also stimulate the intermediary metabolism, and are thus classified as catalysts.
  4. The fourth group is the immunomodulators (IPG’s). Potentized Suis organ extracts, nosodes and also venoms of animal origin act as immunomodulators. These substances will induce Th3 cells and through the immunological bystander reaction will be able to modulate the immune response.

Homotoxicological remedies may also be classified according to the way the above is combined. Five groups stand out for use in the United States:

  1. The Indication-based Heel remedies: These are primarily composed of PPG’s and MPG’s and are often indication-based, as, for example, Traumeel and Vertigoheel. They can be used for acute as well as chronic conditions, and depending on the chronicity, can be used from days to weeks to months.
  2. The Homaccords: These are also primarily composed of PPG’s and MPG’s such as Nux vomica-Homaccord (PPG’s) or Phosphor-Homaccord (PPG’s and MPG’s), which are typically present in potency chords, for instance 3X, 6X, 10X, 30X, 200X, and 1000X. The Homaccords are often organotropic, thus they will support the function of a specific organ, and due to the potency chord will have a far-reaching action on the organ. They are often used for a period of time, 12 weeks for instance. Due to their potency chords, they often will not give a first aggravation seen by other homeopathic remedies.
  3. The Injeels, or Heel remedies: These remedies are like a combination of the indication- based Heel remedy and a Homaccord, and are often directed at treating a specific indication affecting an organ system. The individual components which are normally PPG’s and MPG’s also come in shorter potency chords than the above, for instance, in potency chords to 10X and 30X.
  4. The Catalysts: These contain substances of the CPG’s mentioned above occasionally with added potentized vitamins and other co-factors of the intermediary metabolism. The catalysts which come in a compositum also may contain PPG’s, MPG’s, as well as IPG’s. They are added as soon as a condition reaches the matrix phases, in other words, once it affects the ground regulation system, and are employed in cycles, as the compositae.
  5. The Compositae: These remedies have been developed as the ultimate regulators and modulators. They often contain all four PG’s, namely PPG, MPG, CPG and IPG, and therefore lend themselves to treatment of all conditions, but especially to those in the matrix and cellular phases. Sometimes, however, they are also employed in the humoral phases, on an Auto-regulatory system level, like Echinacea compositum, which is an excellent remedy to use in the inflammatory phase. Towards the right of the biological division, they are employed in cycles of five to six weeks, and the result is then assessed as to whether they should be repeated or not. The compositae come in oral vials, and many are also available in oral tablets.

Disease does not arrive on the cellular level overnight. It will start off as dysregulation of the Auto-regulatory system (ARS) and then move to the matrix, or Ground regulation level, (GRS) and lastly will manifest on the cellular level. This will correspond to the humoral, matrix and cellular phases of the six-phase table.

When disease is in the humoral phases, it will often be enough to only employ remedies of the PPG and MPG group, and therefore most Homaccords, Heel remedies and Indication-based Heel remedies will suffice to restore self-regulation in a patient. However, when disease reaches the Ground regulation level, we need to add a catalyst, and when it approaches the cellular level, we need to mostly add an IPG as well. It is important to note that there are Indication-based Heel remedies other than a compositum, for example Gripp-Heel, which contains animal venom, (Lachesis) and therefore will act as an immunomodulator as well. In Lymphomyosot there is a catalyst (Levothyroxin) and an immunomodulator (Aranea diadema). The latter can thus be employed in all the phases of the six-phase table, whereas a remedy such as Engystol (Vincetoxinum and Sulfur, thus PPG, and MPG), will not be sufficient on its own in the matrix and cellular phases. We will then have to add a CPG such as Coenzyme compositum, and for instance, a compositum containing PPG’s, MPG’S, CPG’s and an IPG in some form.

The choice of remedy is thus made on the phase in the six-phase table in which the condition is classified, and on which part of the Auto-regulatory system or the Ground regulation system will need support.

By introducing the vicariation principle into antihomotoxic therapy, Reckeweg pointed out the dynamics of every disease and/or recovery process. The interrelationships which exist between a bio-system and the damaging homotoxins vary continuously during an illness and during the recovery process. The purposeful, self-regulatory forces of the organism usually are retained during illnesses up to and including phases of the six-phase table of homotoxicosis. In contrast, after the Biological Division is crossed, from phase 4 onward, self-regulation and self-recovery is practically no longer possible for the organism. In this case a therapeutic treatment is required to achieve recovery. Following regressive vicariation, a disease often enters phase 2 or 3. This typically requires a change of the antihomotoxic preparation because in phase 2 a symptomatically indicated acute remedy is usually necessary. In phases 2 and 3, which belong to the humoral phases, the self- regulatory capacity of the organism is still present, so that only stimulative medication is required to initiate inflammatory mechanisms, particularly in the matrix. Usually the excretion of the disease occurs via the skin or the mucous membranes. Increased perspiration, sputum, strong formation of urine, light diarrhea and fever are welcome signs of a shift out of the cellular disease phases which indicates an improvement of the basic illness. In the acute phases 1 and 2, composita preparations such as Traumeel or certain Homaccords are preferable.

Tablets

Homeopathic remedies prepared in solid form are in most cases mixed with milk sugar (lactose) and then formed into tablets. Tablets should be placed under the tongue and allowed to dissolve gradually without chewing. This permits the active ingredients to be absorbed through the buccal lining of the mouth, thereby evading the stomach where gastric juices might destroy or inactivate the remedy.

Oral drops and oral vials

Liquid homeopathic mediciations should be retained in the mouth long enough for their contents to be absorbed by the mouth’s buccal lining. As with tablets, the goal is to avoid the stomach’s juices which can inactivate the remedy. Therefore, liquids should not be simply swallowed.

Ointments and gels

Topical preparations offer a direct application to the affected area. Ointments and gels may be applied generously, as necessary, by rubbing gently into the skin.

Nasal sprays

Homeopathic nasal sprays are not associated with side effects or the risk of habituation. To thoroughly coat the nasal membranes, inhale deeply through each nostril after application.

Suppositories

Suppositories offer an alternate route of administration for persons unable to take  medications orally.

Ear drops

For comfort in administering, warm the liquid by holding the vial in a closed hand for one minute.

Eye drops

Homeopathic eye drops are safe for wearers of contact lenses.

Cough syrup

Homeopathic cough syrups help promote expectoration to shorten the duration of the cough.

Injection solution (Rx)

The injection solution offers doctors the ability to inject medication directly into the affected area. Remedies are contained in vials and diluted in an isotonic sodium chloride solution base.

A

Abdominal bloating

Abortion, threatened

Abrasions and burns

Abscesses

Achalasia

Acne rosacea

Acne vulgaris

Acrocyanosis

Addison’s disease*

Adenoid hypertrophy

Adhesions

Adiposis dolorosa

Adnexitis

Adrenal atrophy

Aging, premature

Agoraphobia

Albuminuria

Alcoholism

Allergic symptoms

Alopecia areata

Alveolitis

Alzheimer’s disease

Amenorrhea, functional

Anal fissures

Anaphylaxis*

Anemia*

Angina pectoris*

Angina tonsillaris*

Angina, ulceromembranous

Angioedema

Ankylosing spondylitis

Anorexia

Anosmia

Anthrax

Anxiety, symptoms of

Aphtha (canker sores)

Apophysitis

Appendicitis, chronic*

Arrythmia

Arteriosclerosis*

Arthritis, symptoms of

Arthritis (inflammatory, rheumatoid and traumatic)

Arthritis of the hip

Arthropathy

Ascites

Asthenia, neurocirculatory

Asthma, bronchial*

Asthma, cardiac*

Atelectasis*

Atheroma

Athlete’s heart*

Atrial fibrillation

Atrophic gastritis*

Atrophic rhinitis

Atrophic vaginitis

Attention Deficit Hyperactivity Disorder (ADHD)

B

Back pain (lower)

Bacterial growth, disturbed

Balanitis

Bartholinitis*

Basal cell carcinoma

Basedow’s disease

Bechterew’s disease

Benign prostatic hypertrophy

Biliary colic

Blepharitis

Bone cysts

Bronchiectasis

Bronchitis

Bronchopneumonia*

Brucellosis* (undulant fever)

Bruxism (grinding or clenching of the teeth)

Bulbar paralysis, progressive

Burns, minor

Bursitis

C

Calcaneal spur

Calcium metabolism, symptoms of

Carbuncles (boils)

Carcinoma

Cardiac insufficiency,* latent

Cardiospasm

Cataract, symptoms of

Causalgia

Cellulitis, symptoms of

Cerebral injury

Cerebral ischemia

Cerebral lesions; partial, fetal, in early childhood

Cerebrosclerosis

Cerumen, excessive

Cervical headache

Cervicobrachial syndrome

Chalazion

Character disturbances

Chilblains

Chills, tendency towards

Cholangitis, cholecystitis*

Cholecystitis

Cholelithiasis

Cholesteatoma

Cholesterolaemia

Chondroma

Chondropathy

Chorea minor (Sydenham’s chorea)

Chorioiditis

Chorea minor (Sydenham’s chorea)

Chron’s disease (ileitis and colitis)

Chronic Fatigue Syndrome (CFIDS)

Chronic obstructive pulmonary disease (COPD)

Cicatricial keloids

Cirrhosis hepatis

Claudication, intermittent (Charcot’s disease)

Colic

Colitis

Colitis, ulcerative

Complex regional pain syndrome

Concussion, minor

Collagen-vascular diseases

Collapse

Condylomas

Conjunctivitis

Constipation

Contracture, Dupuytren’s

Convulsions

Corneal opacity

Corns

Coronary diseases

Coronary heart disease

Coronary insufficiency*

Cough, croupous

Coughs

Cradle cap (seborrhea)

Cramps

Croupous coughs

Cushing’s syndrome*

Cystic fibrosis

Cystitis

Cysts

D

Dacryocystitis

Decubitus ulcers

Dedifferentiation Phases

Delirium tremens

Dementia

Dental caries

Depression, endogenous

Depression, exogenous

Depression, reactive

Dermatitis, acute

Dermatomycosis (ringworm of the nails)

Dermatosis, chronic

Desquamation

Diabetes insipidus*

Diabetes mellitus*

Diarrhea

Diphtheria*

Diathesis, exudative (swelling tendencies)

Disc prolapse

Dislocations and sprains

Disturbance of development

Dislocations and sprains

Diverticulitis

Diverticulosis

Dizziness

Douglas’ abscess

Down’s syndrome

Dropsy

Dumping syndrome (post-gastrectomy)

Duodenal ulcer

Duodenitis

Dupuytren’s contracture

Dysentery, symptoms of*

Dyslipidemia (mild to moderate)

Dysmenorrhea

Dyspepsia (gastric indigestion)

Dyspnea, functional

Dyspnoea

Dysuria

Dystonia, vegetative

Dystrophia adiposogenitalis

E

Ecthyma (pyogenic skin infection)

Eczema

Eczema, ani

Eczema, baker’s

Eczema, palpebral

Edema

Ejaculation, premature

Elephantiasis

Emaciation

Emphysema

Empyema*

Empyema of the maxillary sinus

Encephalitis*

Endocarditis*

Enteritis

Enuresis nocturna (bed wetting)

Eosinophilic pneumopathy*

Epicondylitis

Epididymitis*

Epilepsy*

Epileptiform conditions

Epistaxis* (nosebleed)

Epithelioma* (epithelia neoplasm)

Equilibrium disturbances

Erysipelas, symptoms of

Erythema multiforme

Erythrasma, symptoms of

Erysipelas

Esophagitis (See also GERD)

Eustachian tube* blockage (catarrh of the ear)

Exanthema, acute

Exanthema, drug

Excitation, conditions of hyperactivity

Excretory pancreatic failure

Exhaustion

Exophthalmos

Exostosis

Extra systole

Eye irritation

F

Facial paresis

Fat intolerance

Fatigue

Fatty infitration of liver

Fever

Fertility, disturbances of

Fibroma

Fibroma of the breast* (fibrocystic breast disease)

Fibromyalgia

Fibrositis

Fistula

Fistulae

Flatulence (See also Abdominal bloating)

Folliculitis

Focal toxicoses

Flittering scotoma

Frigidity (decreased libido)

Funicular neuralgia

Furuncular otitis

Furunculosis

G

Ganglion cyst

Gangrene, diabetic,* arteriosclerotic*

Gastralgia

Gastritis, acute*

Gastritis, atrophic

Gastritis, chronic*

Gastritis, hyperplastic*

Gastrocardiac syndrome

Gastroduodenitis

Gastroenteritis

Gastro-Esophogeal Reflux Disease (GERD)

Gastro-intestinal hemorrhage*

Geriatric indications

Gingival abscesses*

Gingivitis

Glaucoma*

Glaucoma*

Globus hystericus

Glomerulonephritis*

Glossitis (inflammation of the tongue)

Glucose intolerance

Goiter*

Gonorrhea*

Gout

Granulocytopenia*

Granuloma

H

Haemarthrosis

Haematomas

Haemorrhages

Haemorrhagic diathesis

Haemorrhoids

Halitosis

Hay fever (allergic rhinitis)

Headache

Heartburn (esophagitis)

Heel spur

Helminthiasis*

Hemarthrosis

Hematomas

Hemorrhagic diathesis*

Hemorrhoids

Hepatitis*

Hernias

Herpes, simplex and zoster

Hiccoughs

Hoarseness

Hordeolum

Hydrarthrosis

Hydrocele

Hydronephrosis

Hyperacidity

Hyperemesis

Hyperesthesia

Hyperhidrosis

Hyperlipidemia

Hypermenorrhea

Hyperoxaluria

Hypersalivation

Hypertension*

Hypertensive heart disease

Hyperthermia

Hyperthyroidism

Hypochondriasis

Hypoglycemia*

Hypomenorrhea

Hypophyseal insufficiency*

Hypoplasia mammae

Hypotension

Hypothyroidism

Hypotonia

I

Icterus

Ileitis and jejunitis

Immune deficiency

Impetigo*

Impotence, male

Incontinence, anal

Incontinence, urinary

Indigestion

Inertia, uterine

Infarct

Infection*

Infection, chronic, recurrent

Infectious diseases

Infertility

Inflammation

Influenza

Inner ear deafness*

Inoculation damage (post-vaccination symptoms).

Inoculation damage (prophylaxis of)

Insomnia

Insufficiency of lymph ststem

Intercostal neuralgia

Interdigital mycosis

Intermenstrual pain

Intermittent lameness

Intertrigo

Intestinal colic

Intestinal spasms

Intestinal stasis

Iridocyclitis*

Iron deficiency anemia

Irritable bladder

Irritable bowel syndrome

Iritis

Itch

K

Keloids

Keratitis

Keratoderma

Kidney stones

Korsakoff’s psychosis

L

Lactorrhea

Laryngitis

Lateral pharyngitis

Lateral sclerosis, amyotrophic

Lead poisoning*

Lentigo (liver spots)

Leucopenia

Leukoplakia

Leukorrhea

Lichen planus

Lipid metabolism disturbance

Lipoma

Liver abscess*

Liver damage and cirrhosis of the liver*

Liver overload, functional

Lumbago

Lupus erythematosus*

Lymphadenitis and lymphangitis

Lymphadenopathy

Lymphatism

Lymphedema

Lymphogranuloma, inguinal

M

Malaria

Macular degeneration

Malabsorption

Malaise (See also Exhaustion)

Malignant anthrax*

Mania

Mastitis

Mastodynia

Mastoiditis

Measles

Megacolon

Melancholia

Ménière’s syndrome

Meningeal reactions

Meningism

Meningitis

Menopause

Menorrhagia

Menstrual disorders

Meteorism

Metritis, parametritis

Microsporea

Migraine

Milk secretion (to promote)

Mononucleosis, infectious (glandular fever)

Morning sickness

Motion sickness

Multiple sclerosis*

Mumps

Muscular atrophy

Muscular dystrophy,* progressive

Muscular rheumatism

Myasthenia gravis*

Mycosis

Myelitis

Myocardial infarction* (as auxiliary remedies)

Myocarditis*

Myoma uteri (fibroid uterus)

Myopia

Myotonia

Myositis ossificans

Myxedema*

N

Naevi

Nasal polyps

Nephrolithiasis* (kidney stones)

Nephrosclerosis*

Nephrosis

Nephrotic syndrome*

Nervousness

Nervous system imbalances, autonomic

Neuralgia

Neurasthenia

Neuritis, optical

Neurodermatitis

Neuroma of a stump

Neurocirculatory asthenia

Neurodystrophy

Neurogenic hypotension (orthostatic hypotension).

Neuroma

Neurosis

Neurotic depression

Nevi

Nutritional disorders in infants

O

Obesity (adiposis)

Obstetrics (general)

Oedema

Oedema (swelling)

Olfactory and gustatory senses, disturbance of

Onchomycosis

Onychorrhexis

Oophoritis*

Orchitis

Organic psychosis syndrome

Ostealgia

Osteoarthritis

Osteochondrosis

Osteomalacia

Osteomyelitis*

Osteophytes of the heels

Osteoporosis

Otitis externa

Otitis media, acute*

Otoliths

Otorrhea

Otosclerosis

Ovarian cysts

Ovarian insufficiency

Ovaritis

Oxaluria

Ozena

Ozaena

P

Pain

Pancreatic failure, excretory

Pancreatic fibrosis

Pancreatitis*

Papilloma (of the bladder)

Paradentosis

Paralysis*

Paralysis, general*

Parametritis

Paraplegia*

Paresis

Parkinson’s disease*

Paronychia

Pelvic Inflammatory Disease (P.I.D.)*

Pemphigus

Periarteritis nodosa*

Periartheritis calcaria (myositis ossificans)

Periarthritis, humeroscapular

Periarthritis, scapulohumeral

Pericarditis

Peridontosis

Peripheral vascular disease

Peritonitis

Pertussis*

Peyronie’s disease

Pfeiffer’s disease

Pharyngeal tonsils, hypertrophy of

Pharyngitis

Phlebitis

Phobias

Pityriasis versicolor

Plantar fasciitis

Plethora

Pleurisy*

Pneumoconiosis

Pneumonia*

Pneumopathy, eosinophilic

Polyarthritis

Polyneuritis

Polyneuropathy

Polyposis coli

Polyps

Polyuria

Porphyria*

Precancerous dermatitis

Precancerous state

Premenstrual syndrome

Proctitis, periproctitis

Prolapse, anal and rectal

Prolapsus ani et recti

Prolapsus uteri

Prolapse, uterine

Prostatic hypertrophy, benign

Prostatitis

Pruritus

Psittacosis

Psoriasis

Psychic symptoms

Pulmonary fibrosis

Pulmonary hemorrhage

Pulmonary tuberculosis

Pulpitis

Purpura hemorrhagica

Pyelitis and pyelonephritis*

Pylorospasm

Q

Quinsy*

R

Radiation sickness

Ranula (sublingual cysts)

Reactive depressive syndrome

Recto-uterine abscess*

Reiter’s syndrome

Renal atrophy

Renal edema*

Renal failure*

Renal secretions, disturbance of

Restless leg syndrome

Reticulocytosis

Retinal detachment*

Retinitis

Retinitis pigmentosa*

Retroflexion, uterine

Rhagades

Rheumatoid arthritis

Rhinitis

Rhinitis, acute

Rhinitis, allergic

Rhinophyma

Rhinorrhea

Rickets*

Ringworm of the nails

Roemheld’s syndrome

Rosacea

Rubella

Rubeola morbilli

S

Salpingitis

Scabies

Scarlatina

Schizoid conditions

Sciatica

Scleritis

Scleroderma

Scotoma*

Scrofulosis

Scurvy

Sebacious cyst (adenoma)

Seborrhea

Senile heart*

Sheehan’s syndrome* (pituitary insufficiency)

Shingles

Shock, conditions of, traumatic*

Shoulder-hand syndrome

Sicca syndrome

Siderosis

Silicosis

Simmond’s disease

Sinus-empyema

Sinusitis

Sinusitis, acute

Sinusitis, chronic

Sjögren’s syndrome

Sleep disturbances

Smoker’s lung

Snake bite*

Solar dermatitis

Spasms

Speech disorders

Spinal paralysis, spastic*

Spondylosis

Sports injuries

Sprains

Stasis, venous

Status asthmaticus

Status epilepticus

Stenocardia

Sterility

Stomatitis aphthosa

Strangury

Stress

Stroke* (hemorrhagic or embolic)

Struma

Strumectomy, condition after

Stumps, painful

Styloiditis (radii)

Subluxations*

Sudeck’s atrophy

Sudoresis of the feet (excessive perspiration)

Sunburn

Sunstroke

Suppuration

Sural spasms

Sweat glands, disorders of

Synovitis

T

Tabes dorsalis (tertiary syphilis)

Tachycardia, paroxysmal

Tears, excessive

Telangiectasia

Tendosynovitis

Tendovaginitis

Tenesmus of the G-I tract

Tenesmus of the urinary tract

Tennis elbow

Testicular atrophy

Tetany

Therapeutical damage

Thrombangitis obliterans

Thrombocytopenia

Thrombophlebitis

Thrush (mucocutaneous candidiasis)

Thyrotoxicosis

Thyroidectomy (cervical syndrome)

Thyroiditis

Tibia, pain in

Tic, convulsive

Tinnitus aurium

TMJ Syndrome

Tongue, burning sensation of

Tongue, condition of

Tonsillar hypertrophy

Tonsillitis

Tooth extraction

Torticollis, spasmodic

Toxoplasmosis*

Tracheitis

Travel sickness

Trichinosis

Trigeminal neuralgia

Tuberculosis of the lungs*

Tumor albicans* (fungal ball)

Tumour albus

Typhoid fever*

U

Ulcerative colitis

Ulcers, corneal

Ulcers, crural

Ulcers, duodenal (PUD)

Ulcers, lower leg

Unrest, motor

Urethral stricture

Urethritis, non-specific*

Urinary tract infection, acute

Urinary tract infection, chronic

Urticaria

Uveitis

V

Vaginal atrophy

Vaginitis

Vagotonia

Varicella

Varicose veins

Verrucae (warts)

Vertigo

Vesicular eruptions (in general and on the conjunctiva)

Veterinary medicine

Viral diseases

Vitiligo

Vitreous body, opacity of

Vomiting

W

Warts

Whooping cough

Wounds